C6H3CH3(OH)(C3H7) contains an aromatic carbon ring and is classified as a monoterpenoid phenol. It melts near 0 degrees C and boils approximately 237 C, commonly occuring as the oil of thyme, with many synthetic pathways as well.
CA RVACROL: “If you extract the carvacrol, be prepared to deal with some of its decomposition products.”
A plant that more efficiently metabolizes necessary carbon through the use of a malate or oxaloacetate shuttle mechanism between mesophyll and bundle-sheath cells. Angiosperm species preferring dryer climates, such as maize, are typical members of the C4 pathway, tending to have Krantz anatomy as well.
C4 PLANT: “How can you tell if you’re looking at a C4 plant?
Sites on cell membranes for affixing C3B, a derivative and active form of C3 or one of its decomposition molecules, C3BI or C3D. The receptors have three designations: CR1, CR2, and CR3, with CR1 holdinga utmost importance for phagocytes, B-Lymphocyes, granulocytes, and other mononuclear cells. This receptor aids in immune response and ingestion of complement-bound cells, and may modulate B-cell activation as well.
C3B RECEPTORS: “If the C3B receptors aren’t functioning well, the disease may persist.”
Protein fragments originating from C3 and C5 molecules, respectively, as they convert to enzymatic structure. The fragments can increase vascularpermeability as they are chemotactic for leucocytes, and also induce anaphylactic properties releasing histamine from nearby mast cells.
C3A, C5A: “Since the patient showed increased histamine response, check also for C3A or C5A.”
Molecule counteracting the activity of the C1 complement activatedesterase. Also affects some blood-clotting biological pathways. The molecule may be absent at birth or inactive in a small population, whereas normal presence in the blood limits effects of angioneurotic edema from esterase-generated vasoactive peptides.
C1A INHIBITOR: “This drug is a C1A inhibitor, so don’t mix it with your other prescription.”